No matter if you live in an apartment or house or just want to keep your home free of EMFs, there are a number of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. emf prevention is also possible to use EMF blocker paint to block EMF radiation from reaching your house. Another easy way to protect your home against EMF radiations is to put up a RF shielding canopy. This is a cloth made of net that has EMF shielding. It's used to block EMFs from entering a room. Another alternative is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. This may account for the long-term responses seen in some patients with advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not evident.
The effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. emf prevention was exposed to RF EMF at frequencies similar to that used in humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF might be based on the fact that cancer cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. emf guard suggest that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.
The tumours in those in the group that were unaffected RF EMF, and were fed a standard mouse diet. The tumors of HCCMF HCCMF group were injected with Huh7 cells when they were five-seven weeks old. The tumours were then euthanized in cases of excessive burden.
The tumors of the three groups also displayed different growth curves. The tumours treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, tumors treated with RCF did not show any reduction in size. The difference was significant. The tumours treated with RCF were able to show necrosis, which is common when tumors are that are exposed to RCF. It is possible that the necrosis was due to the lack of oxygen in the more invasive tumors.
In sum, the results indicate the fact that AM EMF exhibits anticancer properties in vitro as well as in live. A number of studies have proven that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF triggers these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the growth of HCC tumours in vivo.