Whether you live in an apartment or house, or you just want to keep your home free of electromagnetic fields There are a variety of ways you can reduce exposure. One of the most effective is to restrict the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from reaching your home. Another way to shield your home against EMF radiation is to use an RF shielding canopy. It is a type made of net which contains EMF shielding. It's used to block EMFs from entering a room. Another option is to get your home fitted with an electrical enclosure. They are referred to as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could explain the long-term effects observed in some patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients isn't clear.
emf blocker on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. block emf were divided into 3 groups. First, the group that was unaffected to RF EMF. Second group members were exposed to RF EMF at frequencies similar to the frequency used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumours was compared to that of RCF. block emf indicated that tumours treated with HCCMF had significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage in the tumour.
The reason for tumour-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled by CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H may have broader implications for the treatment and diagnosis of various cancers.
The tumors in the control group were not exposed EMF from RF, and fed a normal mouse diet. The tumors of those in the HCCMF group were infected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed after they had a high burden.
The tumours in the three groups also showed distinct growth curves. The tumours treated with HCCMF saw a significant decrease in tumour size after 8 weeks. However, the tumors treated with RCF showed no reduction in size. The difference was significant. The tumors treated with RCF had necrosis, which is common when tumors are that are exposed to RCF. It is possible that this necrosis is caused by the lack of oxygen in larger tumours.
In summary, the results indicate an AM-RF EMF is a powerful source of anticancer activity in vitro and in vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting impact on the development of HCC tumors in the vivo.